1Gross A, McDonnell JM, Korsmeyer SJ. Bcl-2 family members and the mitochondria in apoptosis. Genes Dev, 1999,13 : 1899-1911.
2Maruo T,Matsuo H,Samoto T,et al. Effects of progesterone on uter- ine leiomyoma growth and apoptosis. Steroids ,2000,65:585-592.
3Wu X, Blanck A, Olovsson M, et al. Expression of Bcl4, Bcl-x, Mcl- 1 ,Bax and Bak in human uterine leiomyomas and myometrium during the menstrual cycle and after menopause. J Steroid Biochem Mol Bi- o1,2002 ,80 :77 -83.
8Read JC. Bcl-2 and the regulation of programmed cell death [J]. J Cell Biol, 1994, 124 ( 1-2 ) : 1-6.
9Selvakumaran M, Lin HK,Siin RT,et al. The hovelprimary response gene myD118 and the protooncogenes myb, myc and P, cl-2 modulate transforming growth factor Betal-induce apoptosis of myeloid leukemia cells[J]. Mol Cell Biol, 1994,14(4): 2352-2361.
10Rein Ms.Advances in uterine leiomyoma research:the progesterone hypothesis.Environ Health Perspeet,2000,108:791.
2Elmore S. Apoptosis: a review of programmed cell death [ Jl. Toxicol Patho1,2007,35 (4) :495.
3Vaux DL, Cory S, Adems JM,et al. Bcl -2 genes promotes heamopoi- etic cells survival and cooperates with C - myc to immortalize pro - B - cell[J]. Nature, 1998,335 (6189) :440.
4Brooks C, Dong Z. Regulation of mitochondrlal morphological dynamics during apoptosis by Bcl -2 family proteins: a key in Bak[ J~. Cell Cy- cle,2007,6(24) :3043.
