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足细胞损伤机制研究进展 被引量:3

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出处 《中国中西医结合肾病杂志》 2008年第11期1018-1021,共4页 Chinese Journal of Integrated Traditional and Western Nephrology
基金 国家自然科学基金资助项目(No.30500245) 湖北省卫生厅科研基金资助项目(No.NX200510)
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  • 1Abe K,Miyazaki M,Koji T,et al. Enhanced expression of complernent Csa receptor mRNA in human diseased kidney assessed by in sire hybridization. Kidney Int,2001,60( 1 ) : 137 - 146.
  • 2Couser WG,Johnson RJ,Young BA,et al. The effects of soluble recombinant complement receptor 1 on complement mediated experimental glomerulonephritis. J Am Soc Nephrol, 1995, 5 (11) : 1888 - 1894.
  • 3Cunningham PN, Hack BK, Ren G, et al. Glomerular complement regulation is overwhelmed in passive Heymann nephritis. Kidney Int, 2001,60(3) : 900 - 909.
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  • 5Takano Y, Yamauchi K, Hayakawa K, et al. Transcriptional suppression of nephrin in podocytes by macrophages: roles of inflammatory cytokines and involvement of the PI3K/Akt pathway. FEBS Lett,2007,581 (3) :421 - 426.
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  • 8赖小希,丁国华,黄从新,石明,陈铖.p38丝裂原活化蛋白激酶与c-Jun-N末端激酶信号通路反向调控血管紧张素Ⅱ诱导的人足细胞凋亡[J].北京大学学报(医学版),2004,36(2):131-134. 被引量:10
  • 9梁伟,丁国华,贾俊亚,朱吉莉,陈铖.血管紧张素Ⅱ灌注大鼠模型肾小球足细胞损伤观察[J].实用医学杂志,2006,22(5):497-500. 被引量:9
  • 10朱吉莉,丁国华,陈铖,贾俊亚.血管紧张素Ⅱ输注诱导大鼠足突滤孔膜改变增加蛋白尿产生[J].武汉大学学报(医学版),2007,28(1):62-66. 被引量:4

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