1Mor G, Straszewki S, Kamsteeg M, et al. Role of the Fas/Fas ligand system in female eproductive organs: survival and apoptosis [J]. Biothem Pharmacol,2002,64(9) : 1305-1315.
2Walczak H, Miller RE, Arial K, et al. Tumoricidal activity of tumor necrosis factor-related apoptosis inducing ligand in vivo [J]. Nat Med,1999,5(2) : 157-163.
3Sablko-Downes KA, Russell JH, Cross AH. Role of Fas-FasL interactions in the pathogenesis and regulation of autoimmune demyelinationg disease [J] .J Neuroimmunol, 1999,100(1-2) :42-52.
4Moulian N, Bidauh J, Truffauh F, et al. Thymocyte Fas expression is dysregulated in myasthenia gravis patients with anti-acetylcholine receptor antibody [J]. Blood, 1997,89(9) : 3287-3295.
5Gorak-Stolinska P,Truman JP, Kemeny DM, et al. Actrivation-induced cell death of human T-cell subsets is mediated by Fas and granzyme B but is independent of TNF-alpha [ J ]. J Leukoc Biol, 2001,70( 1 ) : 756-766.
6Kawanami S, Mori S, Kikuchi M, et od. Fas and nicotinic acetylcholine receptor in human myasthenic thymus-immunohistochemical study[J].Fukuoka Igaku Zasshi, 1999,90(6):286-294.
7Masunnaga A, Anti T, Yoshitake T, et al. Reduced expression of apoptosis-related antigens in thymuses from patients with myasthenia gravis[J]. Immunol Lett, 1994,39(2) : 169-172.
8Utsugisawa K, Yamagata M, Saitoh K, et al. Fas antigen gene in thymuses from patients with myasthenia gravis [J]. Rinsho Shinkeigaku,1995,35(2) : 121-125.
9Huang DC,Adams JM,Cory S.The comserved N-terminal BH4 domain of Bcl-2 homologues is essential for inlaibiton of apoptosis and interaction with CED-4[J]. EMBO J, 1998,17(4) : 1029-1039.
10Yokoyama T, Tanahashi M, Kobayashi Y, et al. The expression of Bcl-2 family proteins (Bcl-2, Bd-x, Bax, Bak and Bim) in human lymphocytes[J]. Immunol Lett,2002,81(2) : 107-113.
8[2]Wang W, Ostlie NS, Conti-Fine BM, et al. The susceptibility to experimental myasthenia gravis of STAT6-/- and STAT4-/- BALB/c mice suggests a pathogenic role of Th1 cells. J Immunol, 2004, 172:97-103.
9[5]Paas-Rozner M, Dayan M, Pass Y, et al. Oral administration of a dual analog of two myasthenogenic T cell epitopes down-regulates experimental autoimmune myasthenia gravis in mice. Proc Natl Acad Sci USA, 2000, 97:2168-2173.
10[7]Yang H, Kala M, Scott BG, et al. Cathepsin S is required for murine autoimmune myasthenia gravis pathogenesis. J Immunol, 2005, 174:1729-1737.